SPI- ARDMS Ultrasound physics passing the national exam. all about artifacts part 1. For those who wants to pass the SPI- ARDMS national exam, you can email me to buy … Continue Reading →
Basic Principles of Ultrasound Physics and Artifacts As a Point of Care Ultrasound (POCUS) enthusiast, you may dread the term “Ultrasound Physics” and wished there was a simple way on … Continue Reading →
EYE Ultrasound -part 1 Dr.Steve Ramsey, PhD -Public Health MSc-(hon) in Med Ultrasound. Ophthalmology sonography, Eye ultrasound. I started the Eye sonography back in 1984 to 1988 in Windsor … Continue Reading →
Intussusception pediatric sonography Dr.Steve Ramsey, PhD -Public Health MSc-(hon) in Med Ultrasound. Intussusception – Sonography. Nick names : Sandwich sign, target sign psudokidney sign, Donut sign, bullseyes sign, ‘pitchfork’ or … Continue Reading →
Intussusception Intussusception is the invagination (or telescoping) of one part of bowel into another. Most typically it involves the ileum into the caecum however can occur anywhere in the bowel. … Continue Reading →
Greeting from our paranormal zone blog . Why you should advertise with us? Our 180.000 and more readers are diverse from all walk of life , covering wide range of … Continue Reading →
Is There a Side-Effect by Using Medical Ultrasound Regarding Autism? My research paper will be published in the U.K. the journal of obst/gynecol. This year and this is a sample … Continue Reading →
Is There A Side-effect By Using Medical ultrasound on Autism? BY SAAD ISMAIL ( Steve Ramsey), PhD. MSc medical ultrasound. Advances in knowledge and application to patient care: This study … Continue Reading →
MSK ultrasound cases Jan 2021 These are some of the cases I usually gets in a day when I do the MSK, abdominal, small parts sonography. For those who want … Continue Reading →
SAAD RAMZI ISMAIL ,PHD, BIOGRAPHY of dr saad ramzi ismail, his life, work, schooling and research Continue Reading →
Fibroadenomas are common benign (non-cancerous) breast tumors made up of both glandular tissue and stromal (connective) tissue. Fibroadenomas are most common in women in their 20s and 30s, but they can be found in women of any age. They tend to shrink after a woman goes through menopause
A fibroadenoma is usually felt as a lump in the breast which is smooth to the touch and moves easily under the skin.
Fibroadenomas are usually painless, but sometimes they may feel tender or even painful, particularly just before a period.
Most Fibroadenomas are about 1–3cm in size and are called simple Fibroadenomas. When looked at under a microscope, simple Fibroadenomas will look the same all over.
Simple Fibroadenomas do not increase the risk of developing breast cancer in the future.
Some Fibroadenomas are called complex fibroadenoma. When these are looked at under a microscope, some of the cells have different features.
Having a complex fibroadenoma can vary slightly increase the risk of developing breast cancer in the future.
Occasionally, a fibroadenoma can grow to more than 5cm and may be called a giant fibroadenoma. Those found in teenage girls may be called juvenile Fibroadenomas.
It’s not known what causes a fibroadenoma. It’s thought that it probably occurs because of increased sensitivity to the hormone estrogen. Or if the man hold and play with the breast very hard for long time and can injure the fiber tissue.
Breasts are made up of lobules (milk-producing glands) and ducts (tubes that carry milk to the nipple). These are surrounded by glandular, fibrous and fatty tissue. This tissue gives breasts their size and shape.
Fibroadenomas develop from a lobule. The glandular tissue and ducts grow over the lobule and form a solid lump.
Fibroadenomas are often easier to identify in younger women. If you’re in your early 20s or younger, your fibroadenoma may be diagnosed with a breast examination and ultrasound only. However, if there’s any uncertainty about the diagnosis, a core biopsy or FNA will be done. Continue Reading →
Skeletal dysplasias, also known as osteochondrodysplasias, constitute a group of approximately 450 disorders that affect both bone and cartilage. The newest (tenth version) “Nosology and Classification of Genetic Skeletal Disorders” comprises 461 different diseases that are classified into 42 groups based on their clinical, radiographic, and/or molecular phenotypes. Remarkably, pathogenic variants affecting 437 different genes have been found in 425/461 (92%) of these disorders. Many of these disorders result from new (de novo) dominant mutations, and for the autosomal recessive disorders, many occur in families with no history of a skeletal dysplasia.
The prevalence of skeletal dysplasias is estimated to be approximately 2.4 per 10,000 births. Due to high perinatal mortality, the overall prevalence in perinatal deaths is much higher at 9 per 1,000. Although the occurrence of each individual skeletal dysplasia may be rare, as a group they account for a significant number of newborns with congenital anomalies. The presence of a skeletal dysplasia is not always evident at the time of the fetal anatomical survey, and in particular, some non-lethal skeletal dysplasias may only become apparent in the third trimester.
The fetal skeleton develops relatively early, thus the suspicion of a skeletal dysplasia may be possible as early as the first trimester. The appendicular and axial skeleton undergo a programmed pattern of endochondral ossification, whereas the calvarium and portions of the clavicle and pubis ossify via membraneous ossification. Ossification occurs at relatively early gestational ages: the clavicle and mandible at 8 weeks, the appendicular skeleton, ilium, and scapula by 12 weeks, and the metacarpals and metatarsals by 12–16 weeks. The secondary ossification centers become visible later in gestation, beginning with the calcaneus at 20 weeks, the distal femoral epiphysis after 32 weeks, and the proximal tibial epiphysis after 37 weeks. Continue Reading →
Based on the ultrasound findings and SonoSkills pathology checklist analysis I concluded: \
– Complete tear of the long head of the biceps tendon.
– The rotator interval is “empty”. No sign of the long head of the biceps tendon.
– The long head of the biceps tendon has partially retracted. It’s still located in the distal intertubercular groove, probably fixated or stabilized by the tendon’s vinculum (suspensory ligament).
– Minor degree of neovascularization.
The clinical examination findings of the orthopedic surgeon could be confirmed. Furthermore, the surgeon knows that there is a partial retraction of the long head of the biceps tendon, and that no other anatomical structures where involved. This information can help his clinical decision making. Continue Reading →
Based on the ultrasound findings and SonoSkills pathology checklist analysis I concluded: – Full thickness/ partial width tear supraspinatus tendon – Potentially other partial tears: articular sided near anatomical neck, and mid tendon. – Tendinopathy supraspinatus tendon – Mild sign of neovascularization – SASD bursa effusion – Primary impingement of the supraspinatus tendon and SASD bursa with the coracoacromial ligament during active abduction. Continue Reading →
INTRODUCTION TO MSK SONOGRAPHY ,